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158 Available findings have allowed us to speculate that the biological activities of oxidized AAT may depend on the magnitude of AAT oxidation, in a manner similar to that discussed for HSA (Figure 3)

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A previous review has supported the critical role of acylcarnitine in fatty acid metabolism, ketosis and buffering the concentration ratio of acyl-CoA to free CoA in brain metabolism in neurological disorders (Jones et al., 2010)

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