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Cagrilintide mimics and extends these effects through sustained central amylin receptor activation

R.et al (2020)

Voltage-gated sodium channels (VGSCs) have been identified as crucial components in the molecular mechanisms underlying pain signaling pathways

Key physiological features include: Filaggrin gene mutations , leading to a compromised skin barrier and increased transepidermal water loss Th2-skewed immune response , with elevated IL-4, IL-13, and IgE levels Colonization with Staphylococcus aureus , often contributing to inflammation and secondary infection Heightened mast cell activity , promoting pruritus and dermal reactivity This pathophysiology supports a dual treatment approach: barrier repair and inflammation modulation , often in combination with antimicrobial or immunosuppressive strategies

doi:10.1186/gm367