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Therefore there is an unmet need for therapeutics which can result in a clinically meaningful reduction in ICP without side effects which compromise cognition
The response has been remarkable
That is the honest limitation
JNKs, c-Jun N-terminal kinases
For AD specifically, given the well-established effects of GLP-1 RAs on tau hyperphosphorylation, we recommend that future GLP-1 RA clinical trials focus on the tau-positive subgroup of patients
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