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The environment of transferrin deficiency and iron overload could increase the level of ROS and lead to oxidative stress
In fact, NRF2 activation as a clinical target is well established for the treatment of MS since the potent NRF2-activator dimethyl fumarate (DMF) was approved by FDA as first disease-modifying agent in 2013 (Fox et al., 2012
Like, wow, we've come a long way and still not far enough in terms of FDA approval
Sitagliptin was the first agent introduced (in 2006) with other substances having followed soon after
GLP-1R is expressed in the heart, kidneys, liver, and brain, which is why the clinical literature documents cardiovascular, renal, and neuroprotective effects well beyond glycemic control
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