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Although direct clinical evidence for anti-HCC activity in humans is lacking, its ability to attenuate insulin resistance, oxidative stress, and the inflammation-fibrosis cascade may indirectly reduce HCC risk by optimizing the hepatic oncogenic microenvironment
While this improves how cells use glucose and fats, it doesnt directly change appetite signals or gastric emptying the way GLP-1 medications do
Khan UM, et al
The use of L-carnitine in managing these disorders is a topic of debate
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