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18,19 Given the confounding information, there is no established DR screening protocol upon GLP-1RA initiation
KPV has limited long-term data Most KPV evidence comes from laboratory and animal research rather than long-term human studies
Does not address the broader transit slowdown caused by GLP-1 medications
Selenium studies show that selenium increases glutathione peroxidase levels
The neuroprotective potential of GLP-1 has been established in models of neurodegenerative disorders, including Parkinsons disease and Alzheimers disease (AD), with evidence suggesting it can modify disease progression and ameliorate pathology [29,30,31,32]
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