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Dermal toxicity studies of technical polychlorinated biphenyls and fractions thereof in rabbits

Metabolic reprogramming driven by oncogenes allows cancer cells to maintain deregulated cell proliferation, withstand metabolic challenges that are associated with diminished oxygen and nutrients, maintain a dedifferentiated state through alterations in global patterns of gene expression, and corrupt the surrounding microenvironment to actively facilitate tumor growth and dissemination 16,17,18

Under physiological conditions, Nfr2 is bound to kelch-like ECH-associated protein-1 (Keap1) and thereby sequestered in the cytoplasm, whereas under conditions of oxidative stress, Nfr2 dissociates from Keap1, translocates to the nucleus and induces the transcription of antioxidant enzymes

Patient-reported burden of a neuroendocrine tumor (NET) diagnosis: results from the first global survey of patients with NETs

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