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Mitochondrial dysfunction leads to the decrease of NAD + level, resulting in lysosomal acidification and impaired autophagy, and induces ferroptosis, involving the accumulation of iron in lysosomes and lipid peroxidation
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Once the mitochondrial membranes are protected and repaired, the electron transport chain can resume functioning efficiently without leaking excessive free radicals
Youd think using NAD+ to boost NAD+ was the most direct route, but its not so simple
doi:10.1371/journal.pone.0048208 122
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