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7 It remains elusive whether common key molecular mediators exist in the pathophysiology of both SZ and BP, and if they connect shared genetic risks with gross phenotypic manifestations at the anatomical and clinical levels

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Given its essential role in the detoxification of lipid hydroxy peroxides, and its overexpression in most of myeloid malignancies, GPX4 inhibition has emerged as a promising therapeutic strategy to induce ferroptosis as a mean of eradicating myeloid leukemia cells