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Extreme deficits (800-1,000 calories below maintenance) are unnecessary and counterproductive because they increase muscle catabolism and metabolic adaptation

GLP-1RAs can suppress foam cell formation by activating GLP-1R signaling induced autophagy [134], reducing ACAT1 expression/activity [93], as well as inhibiting the PKA/CD36 pathway to inhibit ox-LDL uptake [135]

BPC-157 works by promoting angiogenesis (new blood vessel formation), modulating nitric oxide pathways, and upregulating growth factors like VEGF and EGF to stimulate tissue regeneration at the cellular level

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