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In addition, it had been found that the Nrf2-mediated anti-ferroptosis activity was dependent on the induction of NQO1, heme oxygenase-1 (HO-1), and ferritin heavy chain (FTH1) (Sun et al., 2016)

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Research shows it enhances fibroblast migration to injury sites through the FAK-paxillin pathway, improves collagen fiber organization and cross-linking, promotes angiogenesis to improve nutrient delivery, and reduces inflammatory mediators while preserving beneficial healing signals