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Postbiotics operate through a distinct, complementary mechanism: continuous modulation of gut-derived signaling pathways that influence enteroendocrine function, insulin sensitivity, inflammation, and metabolic adaptation

Cell GLP-1R signaling increases bihormonal insulin + glucagon + islet cells in mice

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Trial objective and outcomes The objective of this trial was to determine whether a 3-week treatment with the GLP-1 RA dulaglutide compared with placebo reduces fluid intake in patients with primary polydipsia