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This epidemiologic shift has prompted renewed interest in systemic prevention strategies that target the biological drivers of colorectal carcinogenesis rather than relying solely on lesion detection

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These findings position Rg3 as a promising broad-spectrum anti-fibrotic agent (97, 98)

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However, the inhibition of -GCS activity by GSH is of physiological significance [7] and is the rate-limiting step in GSH biosynthesis