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As glycine functions as a co-agonist of the NMDAR [82], the enhanced activation of this receptor, leading to excitotoxicity, has been proposed as an important mechanism underlying the pathophysiology of NKH [79, 83]
Research highlights the observed activity of GHK-Cu in protecting lungs from fibrosis following bleomycin therapy, suggesting its potential interest as a chemotherapy adjuvant research subject to investigate increased medication doses without exacerbating side effects
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But with long summers often exceeding 100F (37C) and high humidity, maintaining optimal health can be challenging
In addition, some quinone compounds, such as -LAPachone and aloin, have similar effects on heart protection [182, 183]
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