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Because cagrilintide is not simply another GLP-1 medicine, comparisons can be misleading
TGF-1 impairs the antifibrotic function of fetal placental mesenchymal stem cells (MSCs) by inducing Smad3-dependent autophagy, which promotes lysosomal degradation of hepatocyte growth factor (HGF)
Mechanistically, SIRT1 may promote autophagy directly through its deacetylase activity on autophagy-related proteins, and indirectly through modulating upstream nutrient-sensing pathways such as AMPKmTORC1, depending on the cellular energy status and the availability of nicotinamide adenine dinucleotide + [7, 61, 62]
This choice reflects stability considerations
Ciriolo, Department of Biology, University of Rome Tor Vergata, Via della Ricerca Scientifica, 00133 Rome, Italy e-mail: [email protected] This article was submitted to Experimental Pharmacology and Drug Discovery, a section of the journal Frontiers in Pharmacology
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