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The phosphorylated SMAD2/3 (pSMAD2/3) then forms a complex with SMAD4,[66] which translocates to the nucleus to regulate gene expression, thereby negatively regulating myoblast proliferation,[14] differentiation, and fusion.[67] MSTN signaling can be inhibited by (1) blocking the interaction between the active C-terminal dimer and ActRIIA/B, as seen with anti-MSTN antibodies such as MYO-029 and landogrozumab
However, plaintiffs have argued that these warnings fail to adequately convey the potential severity of the condition or the full range of circumstances under which it can occur
These guidelines are not only about ensuring the medication works as intended, but also about preventing any potential risks
novembre 2023;36(11)
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