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It is known that acute, but not chronic, central GLP-1 receptor activation directly modulates glucose-induced Insulin secretion implicating a direct brain to islet neuronal communication ( On the other hand, chronic GLP-1 activity in -cells increases its own secretion, feeding an autocrine loop that gets overstimulated with the use of exogenous synthetic GLP-1R agonists [(98)

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Bress AP, et al (2024): The Management of Elevated Blood Pressure in the Acute Care Setting: A Scientific Statement From the American Heart Association

Front Cell Neurosci

Recording your pattern allows your provider to recognize if a change is necessary