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It is important to recognize that clinical trials may have limited power to detect small increases in risk for specific rare cancer types, and longer-term observational studies will continue to provide important safety information

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This cascade stabilizes ACSL4 transcripts, elevates ACSL4 protein levels and promotes mitochondrial ROS accumulation, ultimately driving ferroptosis in alveolar epithelial cells during sepsis (82)

A current drawback of intracellular roGFP2-based mechanistic studies is, however, that the mechanisms and structure-function relationships still have to be validated in vitro

tuberculata reduced the damage caused by oxidative stress and cytokines in follow-up studies (Yamada et al., 2022)