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To account for the induction of ALT in the breast cancer cells (21NT and BT474) and non-induction in normal cells (IBR3 and HMEC), it is known that histone H1 is expressed at higher physiological levels in normal tissues and lower levels in breast cancer cells (Scaffidi 2016)
Moreover, these cells were also found to be more resistant to the cellular apoptosis mediated by the paclitaxel
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