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These analyses are very important additions to the aforementioned clinical observations showing the effect of metabolic disorders on ALS/ FTLD
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Abdulmalik et al., demonstrated a significantly increased expression of collagen and sulfated glycosaminoglycans (s-GAG), as well as the expression of tendon-specific genes, in Ex-4-treated hMSC compared to both untreated and insulin-treated controls [14]
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