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Curcumin also inhibited the progression of fibrosis in liver and lung in animal models of chronic inflammatory diseases (24, 25)
The answer is that these approaches are often complementary rather than competitive
GSH depletion-dependent cell death was prevented by selective ferroptosis inhibitors (8 M Fer-1 and 600 nM Lip-1), iron chelator DFO (80 M), as well as autophagic inhibitors Baf-A1 (75 nM) and 3-MA (10 mM)
Antioxidant support is generally a gradual process rather than an instant fix
doi:10.1146/annurev.arplant.59.032607.092811
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