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Specifically, we observed a stacked, filament-like architecture in which only WD40 domains of COP1 in the top layer are accessible, and a substrate-accessible dimeric architecture that is favored upon co-expression of c-Jun or ETS2

Multivalent interactions of the disordered regions of XLF and XRCC4 foster robust cellular NHEJ and drive the formation of ligation-boosting condensates in vitro

It serves as a primary shield against free radicals

Exposure to fatty acids aggregates lipid accumulation and accelerates tubular cell necroptosis

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