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Mechanistically it is an amylin strategy, not a drop-in GLP-1 substitute

Circumventing the Crabtree effect: forcing oxidative phosphorylation (OXPHOS) via galactose medium increases sensitivity of HepG2 cells to the purine derivative kinetin riboside

Second, some (e.g., turmeric-derived EVs, TDEVs) directly exert anti-inflammatory effects by delivering plant-specific metabolites like curcumin, known to regulate inflammatory and oxidative balance [32, 33]

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