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Results MAb 3F52 acts as a selective inhibitor of the GLP-1R The binding properties of mAb 3F52 to GLP-1R were characterized in a competition binding assay using 125 I-GLP-1(7-37)-OH as the tracer on cell membranes expressing the human GLP-1R
cardiometabolic markers
Fluoxetine further enhances NIX (BNIP3L)-mediated mitophagy in the hippocampus of CUMS mice [127, 128] and promotes autophagic flux and mitophagy in astrocytes via a p53-dependent mechanism, facilitating the clearance of damaged mitochondria [101]
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