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393 As a result, GLP-1RAs could mitigate the detrimental effects of hyperlipidemia-induced skeletal defects, leading to an improved prognosis in individuals with OA

NAC (N-acetylcysteine) as precursor: Rather than supplementing glutathione directly, NAC provides the rate-limiting amino acid (cysteine) for endogenous glutathione synthesis

In mouse organoids, the effect of SCFAs was limited to L-cells alone because markers of other endocrine cell types (Sct, Tph, and Gip) remained unchanged

GLP-1 receptor activation improves cell function and survival following induction of endoplasmic reticulum stress

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