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The main reason is that GSH-Au NCs@His does not have the capacity to form an Au-S bond, which greatly reduces the effectiveness of GSH-Au NCs@His to consume GSH, thus protecting tumor cells from ROS oxidative damage (Figure 7C)

Astrocytes maintain their antioxidative functions through two primary mechanisms: the Nrf2keap1antioxidant response element (ARE) pathway and glutathione (GSH) synthesis

374 Exendin-4 has also been shown to stimulate osteoblast activity and mitigate bone loss in an ovariectomized mouse model