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Using this therapy as a tool to treat PAD in patients without diabetes may be a game-changer in terms of reducing morbidity and mortality. Prospective controlled clinical trials are warranted to examine the current impact of GLP-1RA on non-diabetic PAD patients

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First, mechanistic studies should combine single-cell spatial transcriptomics with mitochondrial metabolomics to map ferroptosis pathways in spinal ventral horn motor neurons, microglia, and astrocytes, with emphasis on the coupling between NCOA4-mediated ferritinophagy and SOD1/TDP-43 clearance, thereby establishing a dynamic model linking mitochondrial iron dysregulation, proteotoxic stress, and cell death