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Description

Protocol duration typically runs 8-16 weeks

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Ferroptosis in non-alcoholic fatty liver disease/non-alcoholic fatty liver disease-hepatocellular carcinoma Metabolic changes and hepatocyte lip toxicity caused by the ectopic accumulation of free fatty acids (FFAs) in the liver are considered to be the principal causes of liver injury in patients with NAFLD (39)

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Glutathione peroxidase [PMID: 7329310] Laboratory literature summary: Glutathione peroxidase

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Cisplatin induces the activation of multiple mediators associated with ER stress, including glucose-regulated protein 78 (GRP78), C/EBP homologous protein (CHOP), phosphorylated c-Jun N-terminal kinase (p-JNK), protein kinase R-like ER kinase (PERK), activating transcription factor-6 (ATF-6), and inositol-requiring enzyme-1 (IRE-1) pathways

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