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Carnitine can help the heart adapt to the disease environment via metabolic reprogramming by regulating changes in oxidative metabolism, glycolysis and ATP production in the damaged myocardium [287]
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KLF2 represses adipogenesis by inhibition of PPAR, C/EBP, and SREBP1 expression in 3T3-L1 preadipocytes
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Thus, repeated intravenous L-carnitine supplementation may produce a greater increase the total carnitine concentration in skeletal muscle than oral L-carnitine during the modified fasting period
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