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Lactic acid is elevated in idiopathic pulmonary fibrosis and induces myofibroblast differentiation via pH-dependent activation of transforming growth factor-beta

It activates GHRH receptors on the pituitary, increasing endogenous GH and downstream IGF-1 signaling

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2004), albeit at higher concentrations than primary cells (Raza and John 2015), they do so via caspase-mediated apoptosis, not necrosis (Jaeschke et al

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