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RYGB is characterized by marked alterations in the secretory profile of gut hormones known to influence appetite with consistent findings of higher postprandial concentrations of the anorexigenic glucagon-like peptide 1 (GLP-1), peptide YY (PYY), cholecystokinin (CCK) and lower concentrations of the orexigenic ghrelin after surgery [10,11,12,13]
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