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Fe + drives the Fenton reaction, producing unstable hydroxyl radicals that initiate lipid oxidation of PUFAs, forming conjugated dienes and eventually yielding toxic lipid peroxidation products such as 4-hydroxynonenal (4-HNE) and MDA

Adams KN, Takaki K, Connolly LE, Wiedenhoft H, Winglee K, Humbert O, Edelstein PH, Cosma CL, Ramakrishnan L

Graphical abstract Similar content being viewed by others Introduction As the central hub of eukaryotic energy metabolism, mitochondria produce adenosine triphosphate (ATP) via oxidative phosphorylation, a process not only essential for cellular energy production but also critically involved in regulating cell cycle progression, redox homeostasis, and programmed cell death [1,2,3]