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[2] [13] Selective glucagon receptor activation may promote energy expenditure and reduce hepatic steatosis (fatty liver)
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This dual hypertrophic-and-hyperplastic mechanism is frequently cited as what distinguishes IGF-1 LR3 from single-pathway anabolic compounds in comparative research
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"but I do find it weird that my B12 is nonexistent and yet my RBC, MCV, and MCH are normal." Low B12 can lead to enlarged red blood cells (macrocytosis)
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