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Published preclinical research (20232024) demonstrated that SLU-PP-332 activates ERR-dependent gene programs governing mitochondrial biogenesis, oxidative fiber-type conversion, and fatty-acid oxidation in skeletal muscle the same pathways activated by endurance exercise training leading researchers to describe it as an "exercise mimetic." Studies published in peer-reviewed pharmacology journals have reported improved exercise capacity, metabolic parameters, and cardiac fatty-acid metabolism in animal models, positioning SLU-PP-332 as one of the most closely watched novel compounds in current exercise-physiology and metabolic-syndrome research, given its potential relevance to populations unable to engage in traditional physical training

2 This is an early application of pharmacogenomics (using genetic information to predict medication response) to obesity treatment

3.1.1 Observational epidemiologic study of green tea consumption and risk of AD Moeko Noguchi-Shinohara et al

Based on evidence from completed cardiovascular and kidney outcome trials, organizations like the American Diabetes Association (ADA), the American Association of Clinical Endocrinologists (AACE)/American College of Endocrinology (ACE), the American College of Cardiology (ACC), and Kidney Disease: Improving Global Outcomes (KDIGO) note the glycemic, weight, and organ protective benefits of GLP-1 RAs

Patients who completed Jorie's 12-week program and lost weight often cannot afford or justify continuing at $780+ per month for extended programs