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Collectively, the existing evidence indicates that multiple oncogenic signaling pathways, such as c-Myc, Kras, and HIF-1, directly regulate key processes in glutamine metabolism, thereby driving the tumor dependence on glutamine
GABA/glutamate neuron differentiation imbalance and increased AKT/mTOR signaling in CNTNAP2(-/-) cerebral organoids
Olfson, M., Gerhard, T., Huang, C., Crystal, S
156 ACADSB expression is low in CRC tissues, negatively correlated with the pathological grade of colorectal cancer, and positively associated with the overall survival rate of CRC patients
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