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To test this hypothesis, we employed AMPK siRNA to investigate the effect of l-carnitine on the AMPK/CPT1 pathway in FLS (Fig

Notably, bile acid signaling pathways can also contribute to OA pathology, as indicated by the alleviation of murine OA after suppression of farnesoid X receptor-the receptor for the bile acid glycoursodeoxycholic acid (GUDCA), which upregulates intestinal glucagon-like peptide-1 secretion (Yang et al., 2025)

Among the module membership was GCL, composed of the catalytic GCLC and the modifier GCLM subunit, shown to be upregulated (Fig

InBody scan numbers often lag behind what patients feel physically

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