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FOXO4-DRI may reduce age-related inflammation Senescent cells often release pro-inflammatory factors known as the senescence-associated secretory phenotype (SASP)
HSD17B14, Hydroxysteroid 17-Beta Dehydrogenase 14
5.2 Implications for research The sensitivity analysis shows that one study could be significant enough to change the outcome
Mechanism involves NO system modulation (homeostatic regulationupregulating when deficient, moderating when excessive), VEGF/VEGFR2 upregulation for angiogenesis, FAK-paxillin pathway activation for cell migration (40% increase per Chang et al.), and EGR-1 transcription factor upregulation for growth factor sensitivity
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