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[6] NAD+ augmentation may counteract age-related NAD+ loss caused by overactive NAD-consuming enzymes (such as the NADase CD38), thereby supporting mitochondrial maintenance and metabolic homeostasis

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Animal studies investigating GHK-Cu's effects on neurodegenerative disease models reveal promising research potential that warrants further investigation

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Medical Disclaimer: This article is for informational purposes only and does not constitute medical advice

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