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Targeting tumour metabolism while inducing the functional recovery of immune cells in the TME is key in immunotherapy, especially for highly immunosuppressed pancreatic tumours
doi: 10.3389/fendo.2024.1448394 Received 13 June 2024 Accepted 22 October 2024 Published 13 November 2024 Volume 15 - 2024 Edited by Fred Sinowatz, Ludwig Maximilian University of Munich, Germany Reviewed by Mayank Choubey, NYU Grossmaan Long Island School of Medicine, United States Gao Fei, Beijing University of Chinese Medicine, China Updates Copyright 2024 An, Xie and Gan
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The metabolic plasticity of cancer is not restricted only to cancer cells but extends throughout the TME (184, 185), as seen in CAFs that secrete lactate, lung mesenchymal-derived cells that transfer lipids, suppressing the functioning of NK cells, and arginine-depleting collagen-producing TAMs, leading to the suppression of CD8 + T cell functions
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