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The proposed net effect in experimental systems is a shift in energy substrate utilisation without the hyperglycaemic liability that pure glucagon agonism would carry alone

[6] [7] [10] GPx1 typically uses glutathione (GSH) as the reductant, but when glutathione synthetase (GSS) is, as in brain mitochondria, -glutamylcysteine can serve as the reductant instead

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