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Levels of amyloid plaques in the brain were reduced, and biomarkers for autophagy functionality were improved (Figure 3 for details)
While LR3-specific data are still emerging, these mechanisms indicate possible neuroprotective effects
The FA end products are further degraded through mitochondrial or peroxisomal -oxidation to generate ATP via acyl-CoA synthetase 2
No reported toxicity or adverse reactions from combined use
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