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GIP perhaps makes a greater contribution to the physiological insulin response to meals than GLP-1, but therapeutic development to date has focused on GLP-1, as the insulinotropic effect of GIP is markedly attenuated in Type 2 diabetes (T2D)
This approach offers promise for breast cancer therapy to overcome MDSC-induced immunosuppression (81)
Hemolysis has been clearly associated with emitting proinflammatory signals through releasing DAMPs [224]
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