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Additionally, evidence supports the involvement of oxidative damage in demyelinization and pathogenesis of MS lesions, acting in cooperation with nitric oxide(NO) radicals [113]
Indeed, endogenous GLP-1 and GLP-1R agonists appear to have differential access to the brain due to factors such as stability, transportability, and diffusivity [20]
DPP-4 inhibitors double the circulating concentrations of both GLP-1 and GIP
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3) Angiotensin Receptor Blockers (ARBs, -sartan) Alternative to ACE inhibitors with similar cardiovascular and renal benefits
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