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In patients receiving tirzepatide, the downstream activation of GLP-1 and GIP receptor-mediated molecular pathways, especially in those with chronic pancreatitis, could increase the risk of developing pancreatic cancer [41]
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Across multiple phase III trials, tirzepatide has consistently produced greater reductions in glycated hemoglobin and body weight compared with established GLP 1 receptor agonists and other standard therapies
The classical redox pathways including WNT/-catenin, PI3K/AKT, NRF2, and p53 can respond to oxidant or antioxidant signals and significantly regulate gene transcription ( in vivo evidence supporting the osteogenic potential of NAC is promising, the inconsistency in its effects across different signaling pathways suggests a research gap
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