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Most patients with EGFR -activating mutations (such as deletions in exon 19 and the L858R substitution mutation) initially respond to the first-generation of EGFR TKIs, but acquired resistance will inevitably emerge during clinical treatment, most frequently owing to the secondary T790M mutation within the ATP binding site of the receptor
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& Haak, J
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Why powder formulations exist in controlled settings: Stability: Lyophilisation extends the shelf life of peptide medications, which can degrade in liquid form
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