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In a p48Cre/+-LSL-KrasG12D/+ mouse model, the proliferation of tumor cells was accompanied by a significant increase in ODC activity, and difluoromethylornithine (DMFO) treatment resulted in significant decreases in ODC, ARG1, spermine synthase (SMS), and spermidine synthase (SRM) expression and an increase in OAZ expression, which inhibited tumor progression [275, 276]

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