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Plasmodium falciparum artemisinin resistance: the effect of heme, protein damage, and parasite cell stress response
In recent years, extensive research (Figure 2) has shown that TMAO can activate the inflammasome NOD-like receptor protein 3 (Nlrp3) through different pathways to activate the inflammatory signal pathway, resulting in aggravation of oxidative stress and, in turn, of endothelial dysfunction
The mixture was centrifuged after the addition of 10% trichloroacetic acid
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