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Conclusion: Normal brain GSH in living brain of chronic MA users is consistent with our previous postmortem brain finding and suggests that any oxidative stress caused by MA, at the doses used by our participants, might not be sufficient to cause either a compensatory increase in, or substantial overutilization of, this antioxidant

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There are ways that compounded GLP-1s can interfere with this approach

RESEARCH EVIDENCE For example, Chang et al