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This paper systematically reviews the regulation of GA metabolism and immune responses in the hyperuricemia occurring due to FA metabolic disorders and identifies targets for personalized therapy based on the underlying mechanisms ( Disordered FA metabolism further promotes inflammation, and elevation of the uric acid levels, primarily through high levels of FAs, activates the initiation of inflammation
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The researchers suggest that changes in gut microbiome composition, particularly involving Akkermansia muciniphila and Parabacteroides distasonis , may explain the effects on GLP-1 and other gut-brain signaling molecules
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