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Glutathione suppresses cerebral infarct volume and cell death after ischemic injury: involvement of FOXO3 inactivation and Bcl2 expression
APX activity increased significantly in the JA-primed seedlings at (0 mM Na 2 CO 3 + 10 M JA) level only, relative to that of the control
13,14 Therefore, orally administered glutathione increased PGC-1 through a non-antioxidant property
CRP binds and aggregates oxidized low-density lipoprotein (ox-LDL) and enhances macrophage oxLDL uptake, promoting mitogen-activated protein kinase activation (171) required for foam cell formation (172)
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